SHOCK
(SHOCK) 《休克》
2012年影响因子
2.612
- 2013-08-19•采用高渗溶液对一种挤压伤大鼠模型进行早期液体复苏
- 2013-08-19•Rhoa在调节出血性休克大鼠孤立性淋巴管泵功能方面的作用
- 2013-05-31•使用高渗溶液对挤压伤大鼠模型进行早期液体复苏
- 2013-05-10•纤维蛋白衍生肽Bβ15-42可缓解肝脏缺血/再灌注损伤大鼠模型的肝损伤
- 2013-05-10•缺氧后处理可通过靶向作用于PUMA而抑制内质网应激介导性心肌细胞凋亡
- 2013-05-10•丹酚酸B可保护大鼠肺部微循环免受脂多糖干扰
最新一期
What’s New in Shock? October 2012
Shock. 38(4):335-336, October 2012.
doi: 10.1097/SHK.0b013e31826ea914
Clinical Aspects
Plasma Levels of Mitochondrial DNA in Patients Presenting to the Emergency Department With Sepsis
Shock. 38(4):337-340, October 2012.
doi: 10.1097/SHK.0b013e318266a169
Resuscitation of Traumatic Hemorrhagic Shock Patients With Hypertonic Saline—Without Dextran—Inhibits Neutrophil and Endothelial Cell Activation
Shock. 38(4):341-350, October 2012.
doi: 10.1097/SHK.0b013e3182635aca
Basic Science Aspects
Type 2 Diabetes Mellitus in Mice Aggravates the Renal Impact of Hemorrhagic Shock
Shock. 38(4):351-355, October 2012.
doi: 10.1097/SHK.0b013e318268810f
Palmitoylethanolamide Reduces Early Renal Dysfunction and Injury Caused by Experimental Ischemia and Reperfusion in Mice
Shock. 38(4):356-366, October 2012.
doi: 10.1097/SHK.0b013e318267bbb9
Temperature and Cell-Type Dependency of Sulfide Effects on Mitochondrial Respiration
Shock. 38(4):367-374, October 2012.
doi: 10.1097/SHK.0b013e3182651fe6
Cyclin-Dependent Kinase Inhibition Reduces Lung Damage in a Mouse Model of Ventilator-Induced Lung Injury
Shock. 38(4):375-380, October 2012.
doi: 10.1097/SHK.0b013e3182656e7b
Acid-Induced Acute Lung Injury in Mice is Associated With p44/42 and c-Jun N-Terminal Kinase Activation and Requires the Function of Tumor Necrosis Factor α Receptor I
Shock. 38(4):381-386, October 2012.
doi: 10.1097/SHK.0b013e3182690ea2
Environmental Hypothermia in Porcine Polytrauma and Hemorrhagic Shock is Safe
Shock. 38(4):387-394, October 2012.
doi: 10.1097/SHK.0b013e3182657a21
STAT1-Deficient Mice Are Resistant to Cecal Ligation and Puncture–Induced Septic Shock
Shock. 38(4):395-402, October 2012.
doi: 10.1097/SHK.0b013e318265a2ab
Role of Peroxynitrite in Sepsis-Induced Acute Kidney Injury in an Experimental Model of Sepsis in Rats
Shock. 38(4):403-410, October 2012.
doi: 10.1097/SHK.0b013e31826660f2
Ischemic Postconditioning During Reperfusion Attenuates Intestinal Injury and Mucosal Cell Apoptosis by Inhibiting JAK/STAT Signaling Activation
Shock. 38(4):411-419, October 2012.
doi: 10.1097/SHK.0b013e3182662266
Early Detection of Subclinical Organ Dysfunction by Microdialysis of the Rectus Abdominis Muscle in a Porcine Model of Critical Intra-Abdominal Hypertension
Shock. 38(4):420-428, October 2012.
doi: 10.1097/SHK.0b013e31825ef7e7
Anaphylactic Shock Decreases Cerebral Blood Flow More Than What Would Be Expected From Severe Arterial Hypotension
Shock. 38(4):429-435, October 2012.
doi: 10.1097/SHK.0b013e3182690897
Role of Non-Muscle Myosin Light Chain Kinase in Neutrophil-Mediated Intestinal Barrier Dysfunction During Thermal Injury
Shock. 38(4):436-443, October 2012.
doi: 10.1097/SHK.0b013e318268c731
Letter to the Editor
Effect of Hydrogen Gas on the Survival Rate of Mice Following Global Cerebral Ischemia (Shock 37(6), 645–652, 2012)
Shock. 38(4):444, October 2012.
doi: 10.1097/SHK.0b013e318268deb2
Reply
Shock. 38(4):444-445, October 2012.
doi: 10.1097/01.shk.0000419721.43269.c7
Erratum
Role of Neutrophil Elastase in the Development of Pulmonary Vascular Injury and Septic Shock in Rats: Erratum
Shock. 38(4):446, October 2012.
doi: 10.1097/SHK.0b013e31826e01ec
